I did some infimetric play for shoulders and arms mid-week with Ed Garbe.  I waited until Sunday and did the following WOW (Chest/Back).

Pulldown SS Systems- Jreps 1/3rds

Nautilus Pullover with SS retrofits

MedX Chest Press-included rest-pause after failure

MedX Compound Row with SS cam

Wendy did here every 14th day WOW that was as follows:

MedX Chest Press

SS Systems Pulldown

MedX Overhead Press

MedX Leg Press (followed by TSC abduction/adduction)

This was a fantastic workout and, for whatever reason, the pump was off the chain.  I followed up with a very busy 3-11pm shift.  Today, I am sore, but not systemically fatigued.

This week I received an email from Sid Morris and Bo Railey of Exercise, Inc. in Indianapolis about one of their clients.  I am posting it now in hopes of getting us focused on the stuff that matters most.  Here is the email, followed by a study that supports the role of exercise in improving CHF.  This was something that until very recently was thought to be irreversible.  We in the HIT field had tons of anecdotal experience of this sort of improvement over the years, but once again science is catching up to us.  THIS, ladies and gentleman, is what we are about.

My client, Jim Schliebner, has had three heart attacks and is a war vet.  3 years ago they measured his left ventricle stroke output for his heart (left ventricle ejection fraction)
and it measured at 44.  Which he tells me is low.  Average is 50-55 and above average and looking great is between 60-65.  He has been training with me for exactly two years.
Three months ago the doctors measured his left ventricle output and it measured at 61.  He is very excited about this and credits the strength training for his progress.  He took
a testimonial sheet home today and will bring it back next week.  I thought his story would be a good one for the web site.


Sid Morris

Exercise Inc.
11145 N. Michigan Rd
Zionsville, IN 46077

(317) 873-5700

Exercise training in patients with advanced chronic heart failure (NYHA IIIb) promotes restoration of peripheral vasomotor function, induction of endogenous regeneration, and improvement of left ventricular function.

Source

Department of Internal Medicine/Cardiology, University of Leipzig-Heart Center, Leipzig, Germany. Sandra.Erbs@medizin.uni-leipzig.de

Abstract

BACKGROUND:

Attenuated peripheral perfusion in patients with advanced chronic heart failure (CHF) is partially the result of endothelial dysfunction. This has been causally linked to an impaired endogenous regenerative capacity of circulating progenitor cells (CPC). The aim of this study was to elucidate whether exercise training (ET) affects exercise intolerance and left ventricular (LV) performance in patients with advancedCHF (New York Heart Association class IIIb) and whether this is associated with correction of peripheral vasomotion and induction of endogenous regeneration.

METHODS AND RESULTS:

Thirty-seven patients with CHF (LV ejection fraction 24+/-2%) were randomly assigned to 12 weeks of ET or sedentary lifestyle (control). At the beginning of the study and after 12 weeks, maximal oxygen consumption (Vo(2)max) and LV ejection fraction were determined; the number of CD34(+)/KDR(+) CPCs was quantified by flow cytometry and CPC functional capacity was determined by migration assay. Flow-mediated dilation was assessed by ultrasound. Capillary density was measured in skeletal muscle tissue samples. In advanced CHF, ET improved Vo(2)max by +2.7+/-2.2 versus -0.8+/-3.1 mL/min/kg in control (P=0.009) and LV ejection fraction by +9.4+/-6.1 versus -0.8+/-5.2% in control (P<0.001). Flow-mediated dilation improved by +7.43+/-2.28 versus +0.09+/-2.18% in control (P<0.001). ET increased the number of CPC by +83+/-60 versus -6+/-109 cells/mL in control (P=0.014) and their migratory capacity by +224+/-263 versus -12+/-159 CPC/1000 plated CPC in control (P=0.03). Skeletal muscle capillary density increased by +0.22+/-0.10 versus -0.02+/-0.16 capillaries per fiber in control (P<0.001).

CONCLUSIONS:

Twelve weeks of ET in patients with advanced CHF is associated with augmented regenerative capacity of CPCs, enhanced flow-mediated dilation suggestive of improvement in endothelial function, skeletal muscle neovascularization, and improved LV function. Clinical Trial Registration- http://www.clinicaltrials.gov. Unique Identifier: NCT00176384.

Imagine if this study could be repeated using a truly meaningful exercise protocol.

Post your WOW’s and your thoughts.